Member Bibliography/Bibliografía de Miembros
This page collects all the personal publications that CienciaPR members have uploaded to their profiles. You can also see all of the authors collected in these publications or all the keywords contained in these publications.
] “AKAP150 contributes to enhanced vascular tone by facilitating large-conductance Ca2+-activated K+ channel remodeling in hyperglycemia and diabetes mellitus.”, Circ Res, vol. 114, no. 4, pp. 607-15, 2014.
“AKAP5 keeps L-type channels and NFAT on their toes.”, Cell Rep, vol. 7, no. 5, pp. 1341-2, 2014.
“Local control of TRPV4 channels by AKAP150-targeted PKC in arterial smooth muscle.”, J Gen Physiol, vol. 143, no. 5, pp. 559-75, 2014.
“Calcium dynamics in vascular smooth muscle.”, Microcirculation, vol. 20, no. 4, pp. 281-9, 2013.
“Capturing single L-type Ca(2+) channel function with optics.”, Biochim Biophys Acta, vol. 1833, no. 7, pp. 1657-64, 2013.
“CaV1.2 sparklets in heart and vascular smooth muscle.”, J Mol Cell Cardiol, vol. 58, pp. 67-76, 2013.
“Local regulation of L-type Ca²⁺ channel sparklets in arterial smooth muscle.”, Microcirculation, vol. 20, no. 4, pp. 290-8, 2013.
“Regulation of L-type calcium channel sparklet activity by c-Src and PKC-α.”, Am J Physiol Cell Physiol, vol. 305, no. 5, pp. C568-77, 2013.
“Anchored phosphatases modulate glucose homeostasis.”, EMBO J, vol. 31, no. 20, pp. 3991-4004, 2012.
“Ca2+ signaling amplification by oligomerization of L-type Cav1.2 channels.”, Proc Natl Acad Sci U S A, vol. 109, no. 5, pp. 1749-54, 2012.
“Relationship between Ca2+ sparklets and sarcoplasmic reticulum Ca2+ load and release in rat cerebral arterial smooth muscle.”, Am J Physiol Heart Circ Physiol, vol. 301, no. 6, pp. H2285-94, 2011.
“Restoration of normal L-type Ca2+ channel function during Timothy syndrome by ablation of an anchoring protein.”, Circ Res, vol. 109, no. 3, pp. 255-61, 2011.
“Elevated Ca2+ sparklet activity during acute hyperglycemia and diabetes in cerebral arterial smooth muscle cells.”, Am J Physiol Cell Physiol, vol. 298, no. 2, pp. C211-20, 2010.
“Increased coupled gating of L-type Ca2+ channels during hypertension and Timothy syndrome.”, Circ Res, vol. 106, no. 4, pp. 748-56, 2010.
“Knockout of Na+/Ca2+ exchanger in smooth muscle attenuates vasoconstriction and L-type Ca2+ channel current and lowers blood pressure.”, Am J Physiol Heart Circ Physiol, vol. 298, no. 5, pp. H1472-83, 2010.
“Natural inequalities: why some L-type Ca2+ channels work harder than others.”, J Gen Physiol, vol. 136, no. 2, pp. 143-7, 2010.
“Sympathetic stimulation of adult cardiomyocytes requires association of AKAP5 with a subpopulation of L-type calcium channels.”, Circ Res, vol. 107, no. 6, pp. 747-56, 2010.
“Molecular and biophysical mechanisms of Ca2+ sparklets in smooth muscle.”, J Mol Cell Cardiol, vol. 47, no. 4, pp. 436-44, 2009.
“AKAP150 is required for stuttering persistent Ca2+ sparklets and angiotensin II-induced hypertension.”, Circ Res, vol. 102, no. 2, pp. e1-e11, 2008.
“Calcium sparklets in arterial smooth muscle.”, Clin Exp Pharmacol Physiol, vol. 35, no. 9, pp. 1121-6, 2008.
“The control of Ca2+ influx and NFATc3 signaling in arterial smooth muscle during hypertension.”, Proc Natl Acad Sci U S A, vol. 105, no. 40, pp. 15623-8, 2008.
“Functional contribution of alpha3L8' to the neuronal nicotinic alpha3 receptor.”, J Neurosci Res, vol. 86, no. 13, pp. 2884-94, 2008.
“Calcium sparklets regulate local and global calcium in murine arterial smooth muscle.”, J Physiol, vol. 579, no. Pt 1, pp. 187-201, 2007.
“Ca(v)1.3 channels produce persistent calcium sparklets, but Ca(v)1.2 channels are responsible for sparklets in mouse arterial smooth muscle.”, Am J Physiol Heart Circ Physiol, vol. 293, no. 3, pp. H1359-70, 2007.
“Contribution of valine 7' of TMD2 to gating of neuronal alpha3 receptor subtypes.”, J Neurosci Res, vol. 84, no. 8, pp. 1778-88, 2006.
