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On this page you can display your personal bibliography. To do this do a search of your own publications in PubMed, Google Scholar, or your favorite literature search or management tool. Import all your publications at once using the "Import My Publications" button, or add them one by one by pressing "Add a publication." To import all publications at once, you must use have a BibTex, EndNote, RIS, MARC, XML file of your publications or a list of PubMed IDs.
“Transgenic mouse model reveals an unsuspected role of the acetylcholine receptor in statin-induced neuromuscular adverse drug reactions.”, Pharmacogenomics J, vol. 13, no. 4, pp. 362-8, 2013.
, “SorLA in glia: shared subcellular distribution patterns with caveolin-1.”, Cell Mol Neurobiol, vol. 32, no. 3, pp. 409-21, 2012.
“Transgenic mouse model reveals an unsuspected role of the acetylcholine receptor in statin-induced neuromuscular adverse drug reactions.”, Pharmacogenomics J, 2012. grajalesreyes2012.pdf (454.92 KB)
, “Decoding pathogenesis of slow-channel congenital myasthenic syndromes using recombinant expression and mice models.”, P R Health Sci J, vol. 29, no. 1, pp. 4-17, 2010.
, “Fourier transform coupled to tryptophan-scanning mutagenesis: lessons from its application to the prediction of secondary structure in the acetylcholine receptor lipid-exposed transmembrane domains.”, Biochim Biophys Acta, vol. 1784, no. 9, pp. 1200-7, 2008.
, “Potential role of caveolin-1-positive domains in the regulation of the acetylcholine receptor's activatable pool: implications in the pathogenesis of a novel congenital myasthenic syndrome.”, Channels (Austin), vol. 2, no. 3, pp. 180-90, 2008.
“Tryptophan-scanning mutagenesis in the alphaM3 transmembrane domain of the muscle-type acetylcholine receptor. A spring model revealed.”, J Biol Chem, vol. 282, no. 12, pp. 9162-71, 2007.
, “Novel beta subunit mutation causes a slow-channel syndrome by enhancing activation and decreasing the rate of agonist dissociation.”, Mol Cell Neurosci, vol. 32, no. 1-2, pp. 82-90, 2006.