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On this page you can display your personal bibliography. To do this do a search of your own publications in PubMed, Google Scholar, or your favorite literature search or management tool. Import all your publications at once using the "Import My Publications" button, or add them one by one by pressing "Add a publication." To import all publications at once, you must use have a BibTex, EndNote, RIS, MARC, XML file of your publications or a list of PubMed IDs.
“SorLA in glia: shared subcellular distribution patterns with caveolin-1.”, Cell Mol Neurobiol, vol. 32, no. 3, pp. 409-21, 2012.
“Tryptophan scanning mutagenesis reveals distortions in the helical structure of the δM4 transmembrane domain of the Torpedo californica nicotinic acetylcholine receptor.”, Channels (Austin), vol. 6, no. 2, pp. 111-23, 2012.
, “Fourier transform coupled tryptophan scanning mutagenesis identifies a bending point on the lipid-exposed δM3 transmembrane domain of the Torpedo californica nicotinic acetylcholine receptor.”, Channels (Austin), vol. 5, no. 4, pp. 345-56, 2011.
, “Decoding pathogenesis of slow-channel congenital myasthenic syndromes using recombinant expression and mice models.”, P R Health Sci J, vol. 29, no. 1, pp. 4-17, 2010.
, “Fourier transform coupled to tryptophan-scanning mutagenesis: lessons from its application to the prediction of secondary structure in the acetylcholine receptor lipid-exposed transmembrane domains.”, Biochim Biophys Acta, vol. 1784, no. 9, pp. 1200-7, 2008.
, “Functional contribution of alpha3L8' to the neuronal nicotinic alpha3 receptor.”, J Neurosci Res, vol. 86, no. 13, pp. 2884-94, 2008.
“Potential role of caveolin-1-positive domains in the regulation of the acetylcholine receptor's activatable pool: implications in the pathogenesis of a novel congenital myasthenic syndrome.”, Channels (Austin), vol. 2, no. 3, pp. 180-90, 2008.
“Tryptophan scanning of the acetylcholine receptor's betaM4 transmembrane domain: decoding allosteric linkage at the lipid-protein interface with ion-channel gating.”, Channels (Austin), vol. 2, no. 6, pp. 439-48, 2008.
, “Tryptophan-scanning mutagenesis in the alphaM3 transmembrane domain of the muscle-type acetylcholine receptor. A spring model revealed.”, J Biol Chem, vol. 282, no. 12, pp. 9162-71, 2007.
, “Contribution of valine 7' of TMD2 to gating of neuronal alpha3 receptor subtypes.”, J Neurosci Res, vol. 84, no. 8, pp. 1778-88, 2006.
“Novel beta subunit mutation causes a slow-channel syndrome by enhancing activation and decreasing the rate of agonist dissociation.”, Mol Cell Neurosci, vol. 32, no. 1-2, pp. 82-90, 2006.
“Tryptophan substitutions reveal the role of nicotinic acetylcholine receptor alpha-TM3 domain in channel gating: differences between Torpedo and muscle-type AChR.”, Biochemistry, vol. 43, no. 1, pp. 78-84, 2004.