Member Bibliography/Bibliografía de Miembros
This page collects all the personal publications that CienciaPR members have uploaded to their profiles. You can also see all of the authors collected in these publications or all the keywords contained in these publications.
Found 27 results
Author Title Type [ Year] Filters: Keyword is Rats, Sprague-Dawley and Author is Quirk, Gregory J [Clear All Filters]
“Revisiting the role of infralimbic cortex in fear extinction with optogenetics.”, J Neurosci, vol. 35, no. 8, pp. 3607-15, 2015.
, “A temporal shift in the circuits mediating retrieval of fear memory.”, Nature, vol. 519, no. 7544, pp. 460-3, 2015.
, “Neural structures mediating expression and extinction of platform-mediated avoidance.”, J Neurosci, vol. 34, no. 29, pp. 9736-42, 2014.
“Fear signaling in the prelimbic-amygdala circuit: a computational modeling and recording study.”, J Neurophysiol, vol. 110, no. 4, pp. 844-61, 2013.
“Prelimbic and infralimbic neurons signal distinct aspects of appetitive instrumental behavior.”, PLoS One, vol. 8, no. 2, p. e57575, 2013.
“Deep brain stimulation of the ventral striatum enhances extinction of conditioned fear.”, Proc Natl Acad Sci U S A, vol. 109, no. 22, pp. 8764-9, 2012.
“Gating of fear in prelimbic cortex by hippocampal and amygdala inputs.”, Neuron, vol. 76, no. 4, pp. 804-12, 2012.
“A time-dependent role of midline thalamic nuclei in the retrieval of fear memory.”, Neuropharmacology, vol. 62, no. 1, pp. 457-63, 2012.
“Dissociable roles of prelimbic and infralimbic cortices, ventral hippocampus, and basolateral amygdala in the expression and extinction of conditioned fear.”, Neuropsychopharmacology, vol. 36, no. 2, pp. 529-38, 2011.
“Memory for fear extinction requires mGluR5-mediated activation of infralimbic neurons.”, Cereb Cortex, vol. 21, no. 3, pp. 727-35, 2011.
“Induction of fear extinction with hippocampal-infralimbic BDNF.”, Science, vol. 328, no. 5983, pp. 1288-90, 2010.
“Infralimbic D2 receptors are necessary for fear extinction and extinction-related tone responses.”, Biol Psychiatry, vol. 68, no. 11, pp. 1055-60, 2010.
“The effects of yohimbine and amphetamine on fear expression and extinction in rats.”, Psychopharmacology (Berl), vol. 204, no. 4, pp. 599-606, 2009.
“Sustained conditioned responses in prelimbic prefrontal neurons are correlated with fear expression and extinction failure.”, J Neurosci, vol. 29, no. 26, pp. 8474-82, 2009.
“Systemic propranolol acts centrally to reduce conditioned fear in rats without impairing extinction.”, Biol Psychiatry, vol. 65, no. 10, pp. 887-92, 2009.
“Fear conditioning and extinction differentially modify the intrinsic excitability of infralimbic neurons.”, J Neurosci, vol. 28, no. 15, pp. 4028-36, 2008.
“Noradrenergic signaling in infralimbic cortex increases cell excitability and strengthens memory for fear extinction.”, J Neurosci, vol. 28, no. 2, pp. 369-75, 2008.
“Activity in prelimbic cortex is necessary for the expression of learned, but not innate, fears.”, J Neurosci, vol. 27, no. 4, pp. 840-4, 2007.
“Consolidation of fear extinction requires NMDA receptor-dependent bursting in the ventromedial prefrontal cortex.”, Neuron, vol. 53, no. 6, pp. 871-80, 2007.
“Inactivation of the ventromedial prefrontal cortex reduces expression of conditioned fear and impairs subsequent recall of extinction.”, Eur J Neurosci, vol. 24, no. 6, pp. 1751-8, 2006.
“Microstimulation reveals opposing influences of prelimbic and infralimbic cortex on the expression of conditioned fear.”, Learn Mem, vol. 13, no. 6, pp. 728-33, 2006.
“Lesions of the basal amygdala block expression of conditioned fear but not extinction.”, J Neurosci, vol. 25, no. 42, pp. 9680-5, 2005.
“Consolidation of fear extinction requires protein synthesis in the medial prefrontal cortex.”, J Neurosci, vol. 24, no. 25, pp. 5704-10, 2004.
“Delayed recall of fear extinction in rats with lesions of ventral medial prefrontal cortex.”, Learn Mem, vol. 11, no. 5, pp. 544-8, 2004.
“Stimulation of medial prefrontal cortex decreases the responsiveness of central amygdala output neurons.”, J Neurosci, vol. 23, no. 25, pp. 8800-7, 2003.