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Post Doctoral Fellow Research Associate at Johns Hopkins University. My undergraduate and graduate studies were performed at the University of Puerto Rico.
RESEARCH EXPERIENCE Johns Hopkins University, Baltimore, MD, Post Doctoral Fellow (Jul. 2006-Present) Principal Investigator: Dr. Alfredo Kirkwood Synaptic Plasticity characterization of Hippocampal CA3 and CA1 circuitry during aging.
University of Puerto Rico, San Juan, Puerto Rico (Aug. 1999- May. 2006) Ph.D. Research Supervisor: Dr. Sandra Peña de Ortiz Dissertation Title: “The role of Genes encoding DNA recombination/repair enzymes in Long-Term Memory Process”.
Cold Spring Harbor Laboratory, Cold Spring Harbor, New York (Summer, 2005) Research Supervisor: Dr. Michael Shen and Dr. Blanche Capel Molecular Embryology of the Mouse Course. Intensive laboratory and lecture course.
University of Puerto Rico, Humacao, Puerto Rico (Aug. 1997- May 1999) Research Supervisor: Dr. Francisco Fuentes Identification of Toluene resistance bacteria’s.
University of Puerto Rico, San Juan, Puerto Rico (Summer, 1998) Research Supervisor: Dr. Sandra Peña de Ortiz Research Title: “Vasopressin gene expression during the learning acquisition of the food search task”.
PUBLICATIONS • M. Colón-Cesario, J. Wang, X.Ramos, H. G. Garcia, J. J. Dávila, J. Laguna, C. Rosado, and S. Peña de Ortiz. (2006) An inhibitor of DNA Recombination Blocks Memory Consolidation, but not Reconsolidation, in Context Fear Conditioning. J. Neurosci. 26: 5524-33.
• S. Peña de Ortiz, M. Colón, and Y. I. Arshavsky. Genomic theory of declarative memory. In Parisi V, De Fonzo V, Aluffi-Pertini F, eds. Dynamical Genetics. Kerala (India): Research Signpost; 2004. pp. 345-364.
• S. Peña de Ortiz, M. Colón, Y. Carrasquillo, B. Padilla and Y. I. Arshavsky. (2003) Experience-dependent neural expression of terminal deoxynucleotidyl transferase in mouse brain. NeuroReport. 14: 1-4.
Synaptic Plasticity characterization of Hippocampal CA3 and CA1 projections during aging. Long-Term Potentiation in CA3 and CA1- Biochemical characterization of the N-methyl-D-aspartic acid (NMDA) receptor and Voltage Dependent Calcium Channels (VDCC) using pharmacological agents. We have demonstrated that Aged rodents used the VDCC biochemical pathway to obtain a successful memory processes, meanwhile Young rodents used the classical NMDA biochemical pathway.
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