Transgenic mouse model reveals an unsuspected role of the acetylcholine receptor in statin-induced neuromuscular adverse drug reactions.

Imagen de Jose Gabriel Grajales Reyes
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TítuloTransgenic mouse model reveals an unsuspected role of the acetylcholine receptor in statin-induced neuromuscular adverse drug reactions.
Publication TypeJournal Article
Year of Publication2013
AutoresGrajales-Reyes, GE, Báez-Pagán, CA, Zhu, H, Grajales-Reyes, JG, Delgado-Vélez, M, García-Beltrán, WF, Luciano, CA, Quesada, O, Ramírez, R, Gómez, CM, Lasalde-Dominicci, JA
JournalPharmacogenomics J
Volume13
Issue4
Pagination362-8
Date Published2013 Aug
ISSN1473-1150
Palabras claveAlleles, Amino Acids, Animals, Caveolin 1, Cholesterol, Drug-Related Side Effects and Adverse Reactions, Gene Expression Regulation, Genome-Wide Association Study, Humans, Mice, Mice, Transgenic, Muscular Diseases, Polymorphism, Single Nucleotide, Receptors, Nicotinic
Abstract

High cholesterol levels are an established risk factor for cardiovascular disease (CVD), the world's leading cause of death. Inhibitors of 3-hydroxy-3-methylglutaryl-coenzyme A reductase (statins) are prescribed to lower serum cholesterol levels and reduce the risk of CVD. Despite the success of statins, many patients abandon treatment owing to neuromuscular adverse drug reactions (ADRs). Genome-wide association studies have identified the single-nucleotide polymorphism (SNP) rs4149056 in the SLCO1B1 gene as being associated with an increased risk for statin-induced ADRs. By studying slow-channel syndrome transgenic mouse models, we determined that statins trigger ADRs in mice expressing the mutant allele of the rs137852808 SNP in the nicotinic acetylcholine receptor (nAChR) α-subunit gene CHRNA1. Mice expressing this allele show a remarkable contamination of end-plates with caveolin-1 and develop early signs of neuromuscular degeneration upon statin treatment. This study demonstrates that genes coding for nAChR subunits may contain variants associated with statin-induced ADRs.

DOI10.1038/tpj.2012.21
Alternate JournalPharmacogenomics J.
PubMed ID22688219
PubMed Central IDPMC3972001
Grant List2R01GM56371-12 / GM / NIGMS NIH HHS / United States
R01 GM056371 / GM / NIGMS NIH HHS / United States
R01 NS033202 / NS / NINDS NIH HHS / United States
R01NS033202 / NS / NINDS NIH HHS / United States
R25 GM061151 / GM / NIGMS NIH HHS / United States
R25GM61151 / GM / NIGMS NIH HHS / United States
T34 GM007821 / GM / NIGMS NIH HHS / United States
T34GM007821 / GM / NIGMS NIH HHS / United States
U54NS0430311 / NS / NINDS NIH HHS / United States

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